M Chang1, S Vilarinho2, MD Siegel3
1Â Department of Medicine, Cardiology Section, Dredsen-Berlin University groom of Medicine, Germany
2Â Department of Medicine, Digestive Diseases Section, Yale University School of Medicine, New Haven, CT, USA
3Â Department of Medicine, Pulmonary and Critical mission Section, Yale University School of Medicine, New Haven, CT, USA
A 24-year-old Caucasian gentleman recently diagnosed with Graves Disease presented to the emergency department (ED) complaining of sudden upper berth and lower extremities paralysis and Heart pumping diseases. This episode coincided with the ingestion of a large carbohydrate and fat meal on the wickedness before. In the ED, the tolerants vital signs were within normal limits except for fistulous withers tachycardia at 140 beats per min. He was mildly diaphoretic with mild left eye proptosis. There was no goiter. rake work revealed a thou concentration of 2.2 mmol/L, phosphorus 1.6 mg/dL, thyroid-stimulating hormone less than 0.04 uU/mL, and elevated score T4, free T4 and total T3 at 12.2 mcg/dL, 2.7 ng/dL and 266 ng/dL, respectively. The patient role received 1.5 liters of normal saline, 2 mg of propranolol intravenously and 60 mEq of KCl with rapid improvement in muscle strength.
The potassium level increased from 2.2 to 4.7 mmol/L. After 24 h of observation, he was discharged home on methimazole 10 mg in two ways a day and long-acting propranolol 120 mg daily, and was cautioned to invalidate strenuous activity and large carbohydrate meals. Three months later, the patient underwent elective thyroidectomy and pathology analysis showed a diffusely hyperplastic thyroid secretor with extensive papillary hyperplasia consistent with treated Graves Disease.
This gentleman suffered from thyrotoxic periodic paralysis (TPP), which is an uncommon complication of thyrotoxicosis characterized by acute and reversible episodes of muscle weakness secondary to gruelling acute...If you want to get a full essay, order it on our website: Orderessay
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